Dompet Transfidelity yang aman & terlindungi
Kendalikan Transfidelity aset Anda sepenuhnya dengan percaya diri di ekosistem Trezor.
- Diamankan oleh dompet perangkat keras Anda
- Gunakan dengan dompet panas yang kompatibel
- Dipercaya oleh lebih dari 2 juta pelanggan

Kirim & terima Transfidelity Anda dengan aplikasi Trezor Suite
Kirim & terima
Dompet perangkat keras Trezor yang mendukung Transfidelity
Sinkronkan Trezor Anda dengan aplikasi dompet
Jaringan Transfidelity yang didukung
Mengapa dompet perangkat keras?
Offline bersama Trezor
- Anda memiliki 100% koin Anda
- Dompet Anda 100% aman secara offline
- Data Anda 100% anonim
- Koin Anda tidak terikat pada perusahaan mana pun
Bursa online
- Jika sebuah bursa gagal, Anda akan kehilangan koin Anda
- Bursa menjadi target para peretas
- Data pribadi Anda mungkin terekspos
- Anda tidak benar-benar memiliki koin Anda
Cara menggunakan FIDEL di Trezor
Hubungkan Trezor Anda
Buka aplikasi dompet pihak ketiga
Kelola aset Anda
Maksimalkan FIDEL Anda
Trezor menjaga FIDEL Anda tetap aman
Dilindungi oleh Secure ElementPerlindungan terbaik terhadap ancaman online dan offline
Token Anda, kendali AndaKontrol penuh atas setiap transaksi dengan konfirmasi langsung di perangkat
Keamanan dimulai dengan open-sourceDesain dompet transparan membuat Trezor Anda lebih baik dan lebih aman
Cadangan dompet yang jelas & sederhanaPulihkan akses ke aset digital Anda dengan standar cadangan baru
Percaya diri sejak hari pertamaSegel pengaman kemasan & perangkat melindungi integritas Trezor Anda
Neurodegenerative diseases such as Alzheimer's Disease are devastating conditions that impact millions globally. These diseases are driven by the misfolding and aggregation of proteins, which lead to brain cell death and cognitive decline. To make matters worse, as we age our cells become more error-prone, resulting in more errors during protein printing, causing proteins to clump together and form aggregates.
Project Transfidelity aims to solve this by improving the accuracy of protein printing, preventing toxic aggregates from forming. Led by Dr. Dimitri Scherbakov and Dr. Rashid Akbergenov, with over 50 years of combined experience in the lab, we have identified two molecules that may improve the efficiency of protein production, potentially preventing protein aggregates, hypothesized to cause dementia.
Despite the significant research funding allocated to neurodegenerative diseases, current therapies continue to treat the symptoms caused by the protein aggregates, with little progress made to stop or reverse disease. We’re trying to go after a potential root cause - rather than trying to clear aggregates once they are there, we aim to prevent them from forming in the first place by reducing the production of faulty proteins. With a clear path towards developing novel intellectual property (IP) and an ever-growing target market, we believe we are poised to make an impact on neurodegenerative disorders and the way we age.